THE LDN-27219 BIOACTIVITY DIARIES

In distinction to neurodegerative disorders and cancer, the contribution of TG2 activity to celiac sprue pathogenesis has long been fairly well established. The deamidation of particular glutamine residues targeted by TG2 in gluten peptides continues to be shown to enhance peptide binding into the sickness-involved proteins HLA-DQ2/eight, which mig